ATC Abstracts

American Transplant Congress abstracts

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Articles tagged "knockout"

  • 2017 American Transplant Congress

    Abrogating MyD88 Signaling in Donor Lung Resident Hematopoietic Cells Promotes Cell Based Tolerance Induction in Lung Allografts in Mice.

    Z. Zheng,1 J. Wang,1 X. Yeap,1 L. Li,3 L. Zhang,2 M. He,1 X. Luo,2 Z. Zhang.1

    1Comprehensive Transplant Center and Department of Surgery, Chicago, IL; 2Division of Nephrology and Hypertension, Chicago, IL; 3Department of Pathology, Chicago, IL

    Objectives: MyD88 signaling plays an important role in allograft rejection and tolerance induction. This study is designed to determine the cellular base of MyD88 expression…
  • 2017 American Transplant Congress

    Deleting LAG-3 Accelerates Cardiac Allograft Rejection and Augments T Cell Memory in Mice.

    J. Erfe,1,2 C. Yang,1 D. Ndishabandi,1 I. Rosales,1 R. White,1 P. Russell,1 R. Colvin,1 J. Madsen,1 A. Alessandrini.1

    1Center for Transplantation Sciences, Massachusetts General Hospital, Boston, MA; 2Yale School of Medicine, New Haven, CT

    Purpose: Lymphocyte Activation Gene-3 (LAG-3) expresses a transmembrane receptor on activated immune cells such as T cells, NK cells, and plasmacytoid dendritic cells. It is…
  • 2017 American Transplant Congress

    Recipient iNOS Deficiency Reduces Spontaneous Kidney Allograft Acceptance by Accelerating Allograft Loss.

    I. Aljabban, B. Jiang, C. Yang, D. Ndishabandi, R. White, P. Russell, J. Madsen, R. Colvin, A. Alessandrini.

    Center for Transplantation Sciences, Massachusetts General Hospital, Boston, MA

    Nitric oxide (NO) production by inducible nitric oxide synthase (iNOS), encoded by NOS2, is a hallmark of the innate immune axis, and is typically associated…
  • 2017 American Transplant Congress

    mTORC1 and 2 Regulate Dendritic Cell (DC) Metabolism and Allostimulatory Function of Donor DCs in Skin Transplantation.

    A. Watson,1,2 H. Dai,1,3 A. Menk,4,5 G. Delgoffe,4,5 A. Thomson.1,2,3

    1Starzl Transplantation Institute, Univ. of Pittsburgh, Pittsburgh; 2Pathology, Univ. of Pittsburgh, Pittsburgh; 3Surgery, Univ. of Pittsburgh, Pittsburgh; 4Immunology, Univ. of Pittsburgh, Pittsburgh; 5Cancer Institute, Univ. of Pittsburgh, Pittsburgh

    Background/Hypothesis: The mechanistic target of rapamycin (mTOR) is a kinase that functions in two complexes: rapamycin (RAPA)-sensitive mTORC1 and RAPA-insensitive mTORC2. These complexes play critical…
  • 2016 American Transplant Congress

    Complement Receptor C5aR2 Deletion in Kidney Transplantation in Mice Attenuates Renal Damage.

    A. Thorenz,1 R. Chen,1 K. Hueper,2 B. Hensen,2 C. Klemann,3 M. Meier,4 A. Klos,5 F. Gueler.1

    1Nephrology, Medical School, Hannover, Germany; 2Radiology, Medical School, Hannover, Germany; 3Paediatric Surgery, Medical School, Hannover, Germany; 4Central Animal Facility, Medical School, Hannover, Germany; 5Microbiology, Medical School, Hannover, Germany.

    ObjectiveThe complement receptors C5aR1 and C5aR2 serve distinct roles in immune regulation. In kidney transplantation the early post-implant ischemia reperfusion injury (IRI) causes rapid complement…
  • 2016 American Transplant Congress

    Genetic TIM-4 Deletion Promotes Immunoregulatory Tissue-Resident Macrophage Survival and Lowers the Barrier to Cardiac Allograft Tolerance.

    T. Thornley,1 P. Kyriazis,1 K. Agarwal,1 Z. Fang,1 L. Ma,1 V. Chipashvili,1 M. Koulmanda,1,2 T. Strom.1,2

    1Medicine, Harvard Medical School and Beth Israel Deaconess Medical Center, Boston, MA; 2Surgery, Harvard Medical School and Beth Israel Deaconess Medical Center, Boston, MA.

    We report that the CD169+TIM-4+ tissue-resident macrophage (TRM) subset is the dominant tissue-resident leukocyte in skin, heart, and pancreas from C57BL/6 mice and skin and…
  • 2016 American Transplant Congress

    Absence of Receptors for C3a or C5a Prevent Alloreactive CD8 T Effector Cell (Teff) Expansion but Differentially Impact Memory (Tmem) Persistence Posttransplantation.

    D. Mathern, P. Heeger.

    Translational Transplant Research Center, Dept of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY.

    As Tmem are known barriers to transplant survival and tolerance in animals and human transplant recipients, understanding factors that drive the formation and survival of…
  • 2016 American Transplant Congress

    Abrogating Donor-Derived Myd88 Signaling Impairs the Recruitment of Recipient Neutrophils and CD8 T Cells into Lung Allografts in Mice.

    Z. Zheng,1 J.-J. Wang,1 X. Yeap,1 X. Kang,1 Y. Xie,1 X. Luo,2 Z. Zhang.1

    1Comprehensive Transplant Center, Northwestern University Feinberg School of Medicine, Chicago, IL; 2Division of Nephrology and Hypertension, Department of Medicine, Northwestern University Feinberg School of Medicine, Chicago, IL.

    Objectives: Transplant ischemia reperfusion injury (IRI) mediated primary graft dysfunction (PGD) remains the predominant cause of short- and long-term graft loss in lung transplantation. This…
  • 2016 American Transplant Congress

    TGFb in Regulatory B Cells-Dependent Tolerance.

    H. Deirawan, L. Kojima, L. Washburn, J. Markmann, J. Kim.

    Surgery, Massachusetts General Hospital, Boston, MA.

    In transplantation, increasing evidence support that B cells can play a major regulatory role in addition to their critical functions in allo-immunity and graft rejection.…
  • 2016 American Transplant Congress

    mTORC2 Negatively Regulates Dendritic Cell (DC) Metabolism and the Allostimulatory Function of Donor DCs in Organ Transplantation.

    A. Watson,1,2 D. Raïche-Regué,1 H. Dai,1 K. Fabian,3,4 G. Delgoffe,4,5 W. Storkus,3,4 A. Thomson.1,2

    1Starzl Transplantation Institute, Univ. of Pittsburgh, Pittsburgh, PA; 2Pathology, Univ. of Pittsburgh, Pittsburgh, PA; 3Dermatology, Univ. of Pittsburgh, Pittsburgh, PA; 4Immunology, Univ. of Pittsburgh, Pittsburgh, PA; 5Cancer Institute, Univ. of Pittsburgh, Pittsburgh, PA.

    The mechanistic target of rapamycin (mTOR) is a kinase that functions in at least two complexes: rapamycin (RAPA)-sensitive mTORC1 and RAPA-insensitive mTORC2. These complexes play…
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