ATC Abstracts

American Transplant Congress abstracts

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Articles tagged "T cell activation"

  • 2017 American Transplant Congress

    Trafficking of Donor Leukocytes and Recipient Cells Cross-Dressed with Donor MHC Molecules After Transplantation of Cardiac Allografts in Monkeys.

    J. Marino, K. Robinson, J. Paster, I. Hanekamp, J. Madsen, G. Benichou.

    Center for Transplantation Sciences, Massachusetts General Hospital / Harvard Medical School, Boston, MA

    Activation of recipient T cells recognizing allogeneic MHC molecules displayed on donor leukocytes is known to trigger acute allograft rejection (direct allorecognition). Recently, we have…
  • 2017 American Transplant Congress

    Preventing of Rejection of Vascularized Composite Allografts by Targeting T Cell Metabolism.

    B. Oh,1 C. Cheng,2 G. Furtmüller,1 W. Lee,1 J. Powell,2 G. Brandacher.1

    1Plastic and Reconstructive Surgery, Johns Hopkins University School of Medicine, Baltimore, MD; 2Oncology, Johns Hopkins University School of Medicine, Baltimore, MD

    [Background] Upon antigen recognition and co-stimulation, T lymphocytes upregulate the metabolic machinery necessary to proliferate and sustain effector function. This metabolic reprogramming in T cells…
  • 2017 American Transplant Congress

    Tofacitinib-Loaded Solid Lipid Nanoparticles Inhibits Dendritic Cell Antigen Presenting Functions and Achieve Selective In Vivo Targeting.

    G. Raimondi,1 X. Calderon-Colon,2 J. Budihardjo,1 A. Mirdad,1 M. Iglesias-Lozano,1 J. Walch,1 W. Lee,1 G. Brandacher,1 J. Patrone.2

    1Plastic & Reconstructive Surgery, Johns Hopkins University, Baltimore; 2Johns Hopkins Applied Physics Laboratory, Laurel

    BackgroundComplications from long-term use of calcineurin inhibitors are a major concern for transplant patients. To develop CNI-free therapies, we investigated an innovative drug delivery method…
  • 2016 American Transplant Congress

    Genetic TIM-4 Deletion Promotes Immunoregulatory Tissue-Resident Macrophage Survival and Lowers the Barrier to Cardiac Allograft Tolerance.

    T. Thornley,1 P. Kyriazis,1 K. Agarwal,1 Z. Fang,1 L. Ma,1 V. Chipashvili,1 M. Koulmanda,1,2 T. Strom.1,2

    1Medicine, Harvard Medical School and Beth Israel Deaconess Medical Center, Boston, MA; 2Surgery, Harvard Medical School and Beth Israel Deaconess Medical Center, Boston, MA.

    We report that the CD169+TIM-4+ tissue-resident macrophage (TRM) subset is the dominant tissue-resident leukocyte in skin, heart, and pancreas from C57BL/6 mice and skin and…
  • 2016 American Transplant Congress

    A P2X7R Mutation with Hypermetabolic T Cells, Th1/Th17 Skewing and Reduced Cardiac Allograft Survival.

    F. D'Addio,1,2 A. Vergani,3 L. Potena,4 A. Maestroni,2 S. Tezza,1 M. Ben Nasr,1 V. Usuelli,2 M. Sbolli,1 L. Borgese,4 A. Iacovoni,5 D. Corradi,6 R. Abdi,7 A. Secchi,2 R. Starling,8 M. Sayegh,7,9 W. Harmon,1 D. Briscoe,1 P. Heeger,10 A. Chandraker,7 F. Grigioni,4 P. Fiorina.1,2

    1Boston Children's Hospital, Boston, MA; 2Ospedale San Raffaele, Milan, Italy; 3Dompe Research, Milan, Italy; 4University of Bologna, Bologna, Italy; 5Ospedale Papa Giovanni XXIII, Bergamo, Italy; 6University of Parma, Parma, Italy; 7Brigham and Women's Hospital, Boston; 8Cleveland Clinic, Cleveland; 9American University of Beirut, Lebanon, Lebanon; 10Icahn School of Medicine at Mount Sinai, New York.

    Background. The purinergic receptor-7 (P2X7R), primarily expressed on lymphocytes, senses ATP released extracellularly during cell damage and regulates T cell activation.Methods. In 640 heart transplant…
  • 2016 American Transplant Congress

    Designing a Novel Highly Selective Immunoproteasome Inhibitor That Promotes Longterm Heart Allograft Acceptance While Inducing Immune Cell Exhaustion, Treg Induction and Memory Cell Suppression.

    E. Sula Karreci,1 A. Mihali,1 M. Uehara,1 A. Kurdi,1 L. Riella,1 R. Abdi,1 H. Fan,2 P. Singh,2 C. Nathan,2 G. Lin,2 J. Azzi.1

    1Transplantation Research Center, Harvard Medical School, Boston, MA; 2Department of Microbiology and Immunology, Weill Cornell Medicine, New York City, NY.

    The constitutive Proteasomes (c-20S) are ubiquitously expressed cellular proteases that degrade polyubiquitinated proteins and regulate cell functions. However, immunoproteasomes (IP or i-20S) are primarily expressed…
  • 2016 American Transplant Congress

    Absence of Receptors for C3a or C5a Prevent Alloreactive CD8 T Effector Cell (Teff) Expansion but Differentially Impact Memory (Tmem) Persistence Posttransplantation.

    D. Mathern, P. Heeger.

    Translational Transplant Research Center, Dept of Medicine, Icahn School of Medicine at Mount Sinai, New York, NY.

    As Tmem are known barriers to transplant survival and tolerance in animals and human transplant recipients, understanding factors that drive the formation and survival of…
  • 2016 American Transplant Congress

    Molecular Significance of Microvascular Inflammation and C4d Negative Transplant Glomerulopathy.

    M. Lubetzky,1 P. O Broin,2 Y. Bao,1 E. Akalin.1

    1Transplantation, Albert Einstein College of Medicine, Bronx; 2School of Mathematics, Statistics & Applied Mathematics, National University of Ireland Galway University, Galway, Ireland.

    Background: Transplant glomerulopathy (TGP) is frequently found in the setting of chronic antibody mediated rejection along with microvascular inflammation (MVI) (peritubular capillaritis+glomerulitis score > 1)…
  • 2016 American Transplant Congress

    Cytometric Deep Profiling of the CD8+ T-Cell Compartment in Transplant Patients with CMV Viremia.

    V. Ferreira, D. Kumar, S. Husain, M. Bhat, P. Ashton, A. Humar.

    UHN, Toronto, Canada.

    Background: Cytomegalovirus (CMV) infection is a common opportunistic viral infection in transplant patients. Patterns of viremia and relapse are influenced by the host immune response,…
  • 2016 American Transplant Congress

    CD57+ CD4 T Cells Persist in the Periphery Following Kidney Transplantation.

    J. Espinosa,1,2 L. Stempora,1 R. Townsend,3 A. Kirk.1

    1Duke University, Durham; 2Emory University, Atlanta; 3Bristol-Meyers Squibb, Princeton.

    Purpose: We have shown that CD57+PD1- CD4 T cells play a role in belatacept-resistant rejection, but their mechanism of action is incompletely defined. Potential mechanisms…
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