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American Transplant Congress abstracts

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Articles tagged "Graft arterlosclerosis"

  • 2020 American Transplant Congress

    Age-Enhanced Transplant Vasculopathy Associates with Impaired Vascular Mitophagy

    M. Blin1, D. Tyrrell1, S. Wood1, D. Farris1, S. G. Tullius2, D. R. Goldstein1

    1Micro and Immunology/Internal Medicine, University of Michigan, Ann Arbor, MI, 2Transplant Surgery, Brigham and Women's Hospital, Boston, MA

    *Purpose: Donor aging leads to increased chronic allograft vasculopathy (CAV) for unclear reasons. Here, we investigated whether impaired alterations in mitophagy, a mechanism to remove…
  • 2020 American Transplant Congress

    Impact of Alemtuzumab Induction on Cardiac Allograft Vasculopathy in Orthotopic Heart Transplant Recipients

    M. Plazak1, X. Gao2, S. Gale2, B. Reed2, R. Madathil1, S. Hammad1, B. Ravichandran1

    1University of Maryland Medical Center, Baltimore, MD, 2University of Maryland School of Pharmacy, Baltimore, MD

    *Purpose: Cardiac allograft vasculopathy (CAV) is impacted by acute rejection and ultimately limits long-term survival of orthotopic heart transplant (OHT) recipients, thus there is interest…
  • 2020 American Transplant Congress

    Impact of Statin Intensity on the Development of Cardiac Allograft Vasculopathy

    K. Goehring1, W. Kuan1, A. Sieg1, K. L. Dawson1, A. R. Kolodziej2

    1Department of Pharmacy, University of Kentucky HealthCare, Lexington, KY, 2Gill Heart and Vascular Institute, University of Kentucky HealthCare, Lexington, KY

    *Purpose: This study aims to examine the efficacy of high-intensity statin therapy compared to low-intensity, moderate-intensity, or no statins at preventing cardiac allograft vasculopathy (CAV)…
  • 2020 American Transplant Congress

    Taking it to the Max: Extremely Low LDL May Provide Benefit for Prevention of Cardiac Allograft Vasculopathy

    R. Lee1, E. J. Henricksen1, Y. Moayedi2, K. Y. Feng3, J. Han3, W. Yang4, R. Imai1, J. J. Teuteberg4, K. K. Khush4

    1Pharmacy, Stanford Health Care, Stanford, CA, 2University Health Network, Toronto, ON, Canada, 3Medicine, Stanford University, Stanford, CA, 4Cardiac Transplant, Stanford University, Stanford, CA

    *Purpose: Routine use of statins is the only Class I ISHLT recommendation in heart transplantation (HT) to reduce cardiac allograft vasculopathy (CAV) and improve survival.…
  • 2020 American Transplant Congress

    Regulatory T Cell-Derived Amphiregulin Activates Fibroblasts to Promote Chronic Rejection after Heart Transplantation

    L. Fan1, X. Zhang1, T. Li1, L. R. Mathews1, G. K. Dwyer2, M. J. Calderon3, K. E. Helfrich3, M. A. Ross3, S. C. Watkins3, H. R. Turnquist1

    1Surgery, University of Pittsburgh, Pittsburgh, PA, 2Immunology, University of Pittsburgh, Pittsburgh, PA, 3Center for Biologic Imaging, University of Pittsburgh, Pittsburgh, PA

    *Purpose: Chronic rejection (CR) after organ transplantation (Tx) manifests as function-limiting allograft fibrosis and vasculopathy. Current immunosuppressants fail to prevent CR and insights into the…
  • 2019 American Transplant Congress

    Identification and Determinants of Cardiac Allograft Vasculopathy Trajectories after Heart Transplantation

    G. Bonnet1, G. Coutance1, M. Racape1, M. Raynaud1, J. Van keer2, M. Bories1, S. Varnous3, P. Rouvier3, R. Guillemain4, L. Lecuyer4, P. Bruneval4, J. Duong-Van-Huyen1, J. Taupin5, M. Naesens2, C. Lefaucheur1, X. Jouven1, A. Loupy1

    1Paris Transplant Group, Paris, France, 2KU Leuven, Leuven, France, 3Pitie Salpetriere Hospital, Paris, France, 4Georges Pompidou Hospital, Paris, France, 5Saint Louis Hospital, Paris, France

    *Purpose: CAV is a major contributor of heart transplant recipient’s mortality. However, little is known about CAV long-term evolution profiles and their determinants. Such information…
  • 2019 American Transplant Congress

    Clonal Composition And Single-cell Characterization Of T-cell Infiltrates In Cardiac Allograft Vasculopathy

    M. V. Habal1, A. Myung2, H. Yan2, S. Rao2, S. Lin2, S. See2, P. Roy2, R. Shihab2, C. Marboe3, S. Restaino1, A. Han2, M. Givertz4, J. C. Madsen5, L. Addonizio1, M. Farr1, E. Zorn2

    1Cardiology, Columbia University Medical Center, New York, NY, 2Columbia Center for Translational Immunology, Columbia University Medical Center, New York, NY, 3Pathology, Columbia University Medical Center, New York, NY, 4Cardiology, Brigham and Women's Hospital, Boston, MA, 5Surgery, Massachusetts General Hospital, Boston, MA

    *Purpose: T-cells are implicated in the pathogenesis of cardiac allograft vasculopathy (CAV), yet their clonal composition and function are incompletely defined. We examined the T-cell…
  • 2019 American Transplant Congress

    Long-Term Influence of Arteriosclerosis in Kidney Grafts Proven by a Zero-Hour Biopsy

    Y. Kakuta1, M. Okumi1, T. Kanzawa1, K. Unagami2, H. Ishida1, K. Tanabe1

    1Urology, Tokyo Women's Medical University, Tokyo, Japan, 2Organ Transplant Medicine, Tokyo Women's Medical University, Tokyo, Japan

    *Purpose: Histopathological assessment of kidney grafts by zero-hour biopsies adds unique information concerning organ quality that is not available on clinical grounds. The most prevalent…
  • 2019 American Transplant Congress

    HbA1c as a Noninvasive Predictor of Chronic Histopathologic Changes in Living Donors for Kidney Transplantation

    K. Tanaka1, S. Nishida1, K. Kinoshita1, Y. Hidaka1, M. Toyoda2, A. Inadome3, A. Takeda4, H. Yokomizo5, S. Uekihara6, J. Shoji7, S. Yamanaga1

    1Surgery, Japanese Red Cross Kumamoto Hospital, Kumamoto, Japan, 2general medicine, Japanese Red Cross Kumamoto Hospital, Kumamoto, Japan, 3Urology, Japanese Red Cross Kumamoto Hospital, Kumamoto, Japan, 4Nephrology, Japanese Red Cross Nagoya Daini Hospital, Nagoya, Japan, 5Surgery, Japanese Red Cross Kumamoto Hospital, kumamoto, Japan, 6general medicine, Japanese Red Cross Kumamoto Hospital, kumamoto, Japan, 7Nephrology, UCSF Connie Frank Transplant Center, San francisco, CA

    *Purpose: The degree of chronic histopathologic changes in renal allografts, including interstitial fibrosis, tubular atrophy, and arteriolar hyalinosis, can have clinical implications in long-term allograft…
  • 2018 American Transplant Congress

    Upregulated IL-33 in Heart Transplants Modulates Local Myeloid Populations to Protect against Chronic Allograft Rejection

    Z. Zhang,1 Q. Liu,1 G. Hussey,2 J. Dziki,2 L. Mathews,1 H. Dai,1 G. Dwyer,1,3 A. Lucas,1 M. Oberbarnscheidt,1 S. Badylak,2 H. Turnquist.1,3

    1University of Pittsburgh, Starzl Transplant Institute, Pittsburgh, PA; 2University of Pittsburgh, McGowan Institute for Regenerative Medicine, Pittsburgh, PA; 3Immunology, University of Pittsburgh, Pittsburgh, PA.

    Purpose: Interleukin-33 (IL-33) is an “alarmin”, or a self-derived molecule that triggers pleotropic immune responses after release from damaged tissue. Yet, how IL-33 shapes alloresponses…
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