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Articles tagged "Nephrotoxicity"

  • 2019 American Transplant Congress

    Eupatilin Ameliorates Tacrolimus Induced Nephrotoxicity via Inhibiting Apoptosis

    H. J. Jang, C. S. Park, M. Y. Oh

    Surgery, University of Ulsan, Gangneung Asan Hospital, Gangneung, Korea, Republic of

    *Purpose: Tacrolimus (FK506) is an immunosuppressant agent that is frequently used to prevent rejection of solid organs upon transplant. However, nephrotoxicity due to apoptosis and…
  • 2018 American Transplant Congress

    Overexpression of BAX, NOL3, and XIAP Apoptotic Genes Participates in Calcineurin Inhibitors Nephrotoxicity: New Insights into Chronic Allograft Dysfunction

    L. Garcia-Covarrubias,1 G. Queipo,2 M. Fonseca,2 D. Alcantara,1 P. Prieto,1 H. Diliz,1 A. Garcia,1 H. Hinojosa,1 A. Cicero,1 G. Melendez,3 M. Alamilla,3 R. Lascurain,4 V. Soto.5

    1Transplantation, Hospital General de Mexico, Mexico City, Mexico; 2Genetics, Hospital General de Mexico, Mexico City, Mexico; 3Research, Hospital General de Mexico, Mexico City, Mexico; 4Faculty of Medicine, UNAM, Mexico City, Mexico; 5Pathology, Hospital General de Mexico, Mexico City, Mexico.

    Introduction: Calcineurin inhibitors toxicity (CNIT) is a factor that limit the outcome of renal transplantation graft. The hypoxia causes histological data as nodular hyalinosis, hyalinosis,…
  • 2018 American Transplant Congress

    Dehydropeptidase-Iinhibitor Reduces Tacrolimus-Induced Kidney Injury by Anti-Oxidative and Anti-Apoptotic Effect

    K. Luo,1 S. Lim,1 J. Il,1 Y. Shin,1 E. Ko,1,2 B. Chung,1,2 H. Hwang,1,2 C. Yang.1,2

    1Seoul St. Mary's Hospital, The Catholic University of Korea, Seoul, Republic of Korea; 2Transplant Research Center, The Catholic University of Korea, Seoul, Republic of Korea.

    Background:Cilastatin is an inhibitor of dehydropeptidase-I which enhances the antibacterial activity of imipenem.Dehydropeptidase I is associated with not only the metabolism of the enzyme but…
  • 2018 American Transplant Congress

    Klotho Enhances FoxO3-Mediated Manganese Superoxide Dismutase Expression by Negatively Regulating PI3K/AKT Pathway during Tacrolimus-Induced Oxidative Stress

    S. Lim,1 K. Luo,1 J. Il,1 Y. Shin,1 E. Ko,1,2 B. Chung,1,2 C. Yang.1,2

    1Transplant Research Center, The Catholic University of Korea, Seoul, Republic of Korea; 2Seoul St. Mary's Hospital, The Catholic University of Korea, Seoul, Republic of Korea.

    Mammalian members of the forkhead box protein O (FoxO) class of transcription factors are implicated in the regulation of oxidative stress, and FoxO proteins are…
  • 2018 American Transplant Congress

    Outcomes in Liver Transplant Patients on Renal Sparing Protocol

    A. Polyak,1 D. Ganger,2 A. Shetty.2

    1New York Medical College, Valhalla, NY; 2Comprehensive Transplant Center, Northwestern University, Chicago, IL.

    As liver transplant outcomes have improved, long term native kidney function has taken a critical role in patient survival. Calcineurin inhibitors (CNIs), such as Tacrolimus…
  • 2017 American Transplant Congress

    Age-Related Differences in Gene Expression Among Kidney Allografts with Normal Histology and Calcineurin Inhibitor Nephrotoxicity.

    E. Rhone,1 S. Bontha,1 P. Walker,2 C. Dumur,3 D. Maluf,1 J. Almenara,3 V. Mas.1

    1University of Virginia, Charlottesville; 2Arkana Laboratories, Little Rock; 3Virginia Commonwealth University, Richmond

    Background Most biomarker research first occurs in adult populations, with consequent extrapolation to children. However, age-related differences may impact gene expression, such as variations in…
  • 2016 American Transplant Congress

    Genomic-Derived Markers to Identify Calcineurin Inhibitor Nephrotoxicity in Pediatric Kidney Transplant Recipients.

    E. Rhone,1 R. Gehrau,2 S. Bontha,2 P. Walker,3 C. Dumur,4 D. Maluf,2 V. Mas.2

    1Pediatrics, University of Virginia, Charlottesville, VA; 2Surgery, University of Virginia, Charlottesville, VA; 3Nephropath, Little Rock, AR; 4Pathology, Virginia Commonwealth University, Richmond, VA.

    Background:Calcineurin inhibitor nephrotoxicity (CNIT) is a well-known contributor to chronic allograft dysfunction in kidney transplant recipients (KTRs). Pediatric KTRs are particularly at risk given their…
  • 2016 American Transplant Congress

    Unexpected Early Calcineurin Inhibitor Nephrotoxicity in Pediatric Kidney Transplant Recipients.

    M. Yanik,1 R. Mannon,2 M. Seifert.1

    1Pediatrics, University of Alabama at Birmingham, Birmingham, AL; 2Medicine, University of Alabama at Birmingham, Birmingham, AL.

    Background: Calcineurin inhibitors effectively suppress the recipient immune response and facilitate early success in kidney transplantation. However, these drugs are potent nephrotoxins and contribute to…
  • 2016 American Transplant Congress

    Early Proteomic Predictors of Late Chronic Kidney Disease in Liver Transplant Recipients.

    J. Levitsky,1 S. Asrani,2 L. Zhao,1 M. Abecassis,1 L. Jennings,2 G. Klintmalm.2

    1Northwestern, Chicago; 2Baylor, Dallas.

    Background: A high proportion of patients develop chronic kidney disease (CKD) after liver transplantation (LT). However, the ability to better select patients for nephroprotective interventions…
  • 2016 American Transplant Congress

    Quadruple Induction Regimen with Basiliximab, Everolimus, Mycophenolate and Very Low Dose Tacrolimus Improves Graft Function without Increase in the Incidence of Microvascular Inflammation or De Novo Donor Specific Antibody Compared to the Standard Triple Regimen.

    M. Miura, H. Higashiyama.

    Department of Urology and Renal Transplantation Surgery, Sapporo Hokuyu Hospital, Sapporo, Japan.

    Objectives: Nephrotoxicity of calcineurin inhibitors (CNI) in kidney allografts has been a significant problem even with low exposure regimens. On the other hand, CNI-free regimen…
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